AUTIZM SPEKTR BUZILISHI: ETIOLOGIK NAZARIYALAR

Authors

  • Mamajanov Taxirjon Gapirjanovich Qo’qon universiteti Andijon filiali Klinik fanlar kafedrasi assistenti Andijon, O’zbekiston Author

DOI:

https://doi.org/10.5281/zenodo.20287438

Keywords:

ning, genetik, lgan, atrof, muhit

Abstract

Autizm spektr buzilishi (ASB) murakkab neyro-rivojlanish holati bo‘lib, uning etiopatogenezi uzoq yillar davomida ilmiy tadqiqotlarning diqqat markazida turibdi. ASB ning kelib chiqishi yagona sababga bog‘liq emas, balki bir necha omillarining o‘zaro murakkab va dinamik ta’siri natijasida yuzaga kelishi tobora ko‘proq empirik dalillar bilan tasdiqlanmoqda [1].

References

Lord, C., Elsabbagh, M., Baird, G., & Veenstra-Vanderweele, J. (2020). Autism spectrum disorder. *The Lancet*, 396(10264), 1836-1849.

Modabbernia, A., Velthorst, E., & Reichenberg, A. (2017). Environmental risk factors for autism: An evidence-based comprehensive review of systematic reviews and meta-analyses. *Molecular Psychiatry*, 22(3), 406-428.

Geschwind, D. H., & State, M. W. (2015). Gene hunting in autism spectrum disorder: on the path to precision medicine. *The Lancet Neurology*, 14(11), 1100-1110.

Sanders, S. J., Sahin, M., & Geschwind, D. H. (2018). The genetics of autism spectrum disorder: a review. *JAMA Psychiatry*, 75(12), 1299-1308.

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Published

2026-05-19

How to Cite

Mamajanov, T. (2026). AUTIZM SPEKTR BUZILISHI: ETIOLOGIK NAZARIYALAR. Science and Innovation, 4(41), 71-73. https://doi.org/10.5281/zenodo.20287438
Innovative Academy RSC
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